Get Chloride Movements Across Cellular Membranes PDF

By Michael Pusch

ISBN-10: 0444528725

ISBN-13: 9780444528728

All residing cells are surrounded by means of a lipidic membrane that isolates them from the customarily harsh atmosphere. despite the fact that, to absorb meals, to excrete waste, and to speak between one another, Nature has invented a very various set of transmembrane shipping proteins. really expert transporters exist to trip electrically charged ions, confident cations like sodium or unfavourable anions like chloride, around the membrane. within the contemporary years, super development has been made within the box of chloride shipping. the current booklet offers the cutting-edge of this quickly increasing and interest-gaining box of membrane shipping. it truly is addressed at a large medically, physiologically, biologically, and biophysically readership. * Describes the state of the art in anion delivery examine* Written through leaders within the box* provides a well timed dialogue of this speedily rising and increasing box

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Michael Pusch's Chloride Movements Across Cellular Membranes PDF

All residing cells are surrounded by means of a lipidic membrane that isolates them from the usually harsh atmosphere. even though, to take in foodstuff, to excrete waste, and to speak between one another, Nature has invented an exceptionally assorted set of transmembrane shipping proteins. really good transporters exist to travel electrically charged ions, confident cations like sodium or unfavourable anions like chloride, around the membrane.

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Extra info for Chloride Movements Across Cellular Membranes

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The disease is due to a hyperexcitability of the muscle cell membrane leading to series of repetitive action potentials, so‐called myotonic runs, that can be detected in electromyograms. Which ClÀ channel mediates the high ClÀ conductance in the muscle membrane? Since the electric organ of rays is developmentally derived from skeletal muscle, it seemed likely that the ClÀ conductance of mammalian muscle might be mediated by a homologue of the Torpedo channel ClC‐0. , 1991b). This channel, which was named ClC‐1, is 54% identical to the Torpedo channel ClC‐0 and represents its closest mammalian orthologue.

The uptake of lactoglobulin is specifically reduced in the ClC‐5‐deficient cells indicating a cell‐autonomous endocytosis defect. (B) The expression of the endocytic receptor megalin was analyzed by confocal immunocytochemistry of kidney slices from heterozygous females. The cells lacking ClC‐5 express less megalin. This result was confirmed by Western blot analysis of megalin in WT, heterozygous, and KO kidneys (inset). Modified from Piwon et al. (2000). far unknown cellular factors. However, this is not the only question remaining unanswered.

Patients with myotonia experience muscle stiffness after voluntary contraction which gradually improves with exercise (“warm‐ up” phenomenon). The disease is due to a hyperexcitability of the muscle cell membrane leading to series of repetitive action potentials, so‐called myotonic runs, that can be detected in electromyograms. Which ClÀ channel mediates the high ClÀ conductance in the muscle membrane? Since the electric organ of rays is developmentally derived from skeletal muscle, it seemed likely that the ClÀ conductance of mammalian muscle might be mediated by a homologue of the Torpedo channel ClC‐0.

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Chloride Movements Across Cellular Membranes by Michael Pusch


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