By Michael Bader
Chapter 1 Glucocorticoids and Mineralocorticoids (pages 1–37): Eilidh Craigie, John J. Mullins and Matthew A. Bailey
Chapter 2 intercourse Steroid Hormones (pages 39–64): Vera Regitz?Zagrosek, Eva Becher, Shokoufeh Mahmoodzadeh and Carola Schubert
Chapter three Angiotensins (pages 65–100): Robson Augusto Souza Dos Santos, Anderson Jose Ferreira and Ana Cristina Simoes e Silva
Chapter four Kinins (pages 101–123): Suzana Macedo de Oliveira, Kely de Picoli Souza, Michael Bader and Joao Bosco Pesquero
Chapter five Natriuretic Peptides (pages 125–141): Paula M. Bryan and Lincoln R. Potter
Chapter 6 Endothelins (pages 143–167): Gian Paolo Rossi and Teresa M. Seccia
Chapter 7 Adrenomedullin (pages 169–191): Istvan Szokodi and Heikki Ruskoaho
Chapter eight Apelin and Vasopressin (pages 193–208): Xavier Iturrioz, Annabelle Reaux?Le Goazigo, Francoise Moos and Catherine Llorens?Cortes
Chapter nine Serotonin (pages 209–231): Prof. Dr. Michael Bader
Chapter 10 Adrenaline and Noradrenaline (pages 233–250): Nadine Beetz and Lutz Hein
Chapter eleven Dopamine (pages 251–293): Pedro Gomes and Patriio Soares?da?silva
Chapter 12 Histamine (pages 295–314): Izabela Rozenberg, Felix C. Tanner and Thomas F. Luscher
Chapter thirteen Prostaglandins and Leukotrienes (pages 315–331): Katharina Lotzer and Andreas J. R. Habenicht
Chapter 14 Cytochrome P450?Dependent Eicosanoids (pages 333–372): Wolf?Hagen Schunck and Cosima Schmidt
Chapter 15 Nucleotides and the Purinergic process (pages 373–393): Vera Jankowski and Joachim Jankowski
Chapter sixteen Nitric Oxide (pages 395–406): Valerie B. Schini?Kerth and Paul M. Vanhoutte
Chapter 17 Acetylcholine (pages 407–424): Maria Claudia Irigoyen, Catarina S. Porto, Pedro Paulo Soares, Fernanda Consolin?Colombo and Antonio Claudio Nobrega
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Extra info for Cardiovascular Hormone Systems: From Molecular Mechanisms to Novel Therapeutics
A more direct relationship between 11βHSD2 and blood pressure was obtained in nonhypertensive individuals subject to salt loading. For those individuals with salt-sensitive blood pressure, the extent of the rise in blood pressure following salt load was indirectly related to 11βHSD2 activity. That is, the lower the activity, the more exaggerated the response to salt. Our preliminary observations support this relationship ﬁnding that mice heterozygote null for hsd11b2 have salt-sensitive blood pressure and an impaired ability to excrete sodium.
This vasodilation results from stimulation of NO production by the endothelium . There is, however, also an effect on the VSMCs to promote vasoconstriction and the net effect on vascular function depends on the balance between the two opposing forces. Thus, inhibition of endothelial NO synthase with N-monomethyl-L-arginine leads to a powerful and sustained vasoconstriction . These observations explain why the effect of aldosterone on vascular tone may vary in different vascular beds. Moreover, local vasodilation may be offset by blunting of the baroreceptor reﬂex  and indirect activation of the sympathetic nervous system.
3 The HPA axis. 9 10 1 Glucocorticoids and Mineralocorticoids V1b receptor  have normal ACTH and corticosterone levels. Such studies do, however, demonstrate a key role for ADH in sustaining the ACTH response to stress. That the HPA axis is not abolished by combined administration of antisera to CRF and AVP – or indeed by double knockout of the receptors – indicates other regulatory factors about which less is known. A number of neuroactive compounds, such as Ang II , catecholamines and glutamate , have been implicated.
Cardiovascular Hormone Systems: From Molecular Mechanisms to Novel Therapeutics by Michael Bader