By John Elefteriades
Masking the pathophysiology, imaging, prognosis, and remedy of various aortic aneurysms and dissections, this resource is helping physicians successfully study and overview affected members in scientific or emergency care settings. delivering a wide range of illustrations, x-rays, and operative pictures to stress key anatomic observations, this consultant includes state-of-the-art perception at the newest biologic, radiologic, scientific, and surgical advancements that experience taken position within the box. awarded in a reader-friendly layout, this resource presents end-of-chapter questions and a point-counterpoint layout to research differing views from well known specialists on those ailments. The Q
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Additional resources for Acute aortic disease
Aortic rupture, after blunt chest trauma, is frequently associated with myocardial contusion, which can lead to cardiac failure, myocardial infarction, and tamponade. Aneurysm formation and aortic rupture can also occur after aortic surgery and even after cardiopulmonary resuscitation (28–30). Extracorporal shock waves for lithotripsy can produce aortic injury as well (31). Another possible cause of trauma is cardiac catheterization, both diagnostic and interventional procedures (32,33). Aortic dissection may be observed in patients who have undergone prior aortic valve replacement, while the interval between valve replacement and dissection varies greatly (34–36).
Indd21 21 2/14/2007 5:51:21 PM 22 Nienaber and Ince 34. Pieters FAA, Widdershoven JW, Gerardy AC, et al. Risk of aortic dissection after aortic valve replacement. Am J Cardiol 1993; 72:1043–1047. 35. Epperlein S, Mohr-Kahaly S, Erbel R, et al. Aorta and aortic valve morphologies predisposing to aortic dissection. An in vivo assessment with transesophageal echocardiography. Eur Heart J 1994; 15:1520–1527. 36. von Kodolitsch Y, Simic O, Schwartz A, et al. Predictors of proximal aortic dissection at the time of aortic valve replacement.
Although straightforward, the Stanford classification (44), based only on the presence (type A) or absence (type B) of false lumen propagation in the ascending aorta, is limited by its simplicity (Fig. 1). Frequent clinically relevant variations in the site of intimal tear cannot be accommodated by these schemes. Often, it is assumed that the intimal tear is above the commissura in type A dissection and in proximity to the subclavian artery in type B dissection; but other clinical patterns are common.
Acute aortic disease by John Elefteriades